Version history
SimShockPad
30
ASO score
Text
31/100
Reviews
0/100
Graphic
60/100
Other
0/100
App Rating
3.7
Votes
7
App Age
6m 7d
Last Update
Dec 11, 2025
Compare with Category Top Apps
|
Metrics
|
Current App
|
Category Top Average
|
Difference
|
|---|---|---|---|
|
Rating
|
3.67
|
4.62
|
-21%
|
|
Number of Ratings (Voted)
|
7
|
66.5K
|
-100%
|
|
App Age
|
0y 6m
|
9y 1m
|
-94%
|
|
In-app Purchases Price
|
$0
|
$31
|
|
|
Update Frequency
|
19d
|
106d
|
-82%
|
|
Title Length
|
11
|
26
|
-58%
|
|
Subtitle Length
|
22
|
26
|
-15%
|
|
Description Length
|
3 315
|
2 631
|
+26%
|
|
Number of Screenshots
|
780
|
1365
|
-43
%
|
|
Size
|
126MB
|
277MB
|
-55
%
|
Category Ranking in United States
7 days
Last 7 days
Last 30 days
Last 90 days
Last 180 days
Last year
| Top | Dec 09, 2025 | Dec 16, 2025 |
|---|---|---|
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No results were found!
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| Top | Dec 09, 2025 | Dec 16, 2025 |
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No results were found!
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| Top | Dec 09, 2025 | Dec 16, 2025 |
|---|---|---|
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No results were found!
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| Top | Dec 09, 2025 | Dec 16, 2025 |
|---|---|---|
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No results were found!
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Ranking Keywords in United States
| Keywords | App Rank |
|---|
Analyze this and other apps using Asolytics tools
Text ASO
Title
(
Characters:
11
of 30
)
SimShockPad
Subtitle
(
Characters:
22
of 30
)
Hemodynamic Simulator.
Description
(
Characters:
3315
of 4000
)
SimShockPad is an interactive emergency simulator designed to train and challenge your skills in managing critical hemodynamic situations. Experience realistic scenarios where the patient may present acute heart failure, major bleeding, or septic shock. Make fast and accurate decisions under pressure to keep the patient alive.
MAIN FEATURES
• Three pathological modes: heart failure, bleeding, and sepsis, with real time parameter changes based on your actions.
• Seven combinable therapies: diuretics, vasopressors, inotropes, nitrites, BIPAP, and transfusion, with dose dependent effects.
• Advanced treatments: correct antibiotic choice for septic focus, transfusion in bleeding, and defibrillation in severe arrhythmias.
• Emergency responses: tachycardia, atrial fibrillation, asystole, and cardiac arrest.
• Multiparameter monitoring: dynamic bars, animated ECG strip, and respiratory wave.
• Game speed options: slow, normal, or fast.
• Adjustable difficulty with three levels.
• Professional multilingual interface in Spanish, English, German, and Chinese.
• Compatible with iPhone, iPad, and macOS.
PATHOLOGICAL MODES
Heart Failure High Type
Severe congestion with very high filling pressures and high vascular resistance. Mean arterial pressure tends to be high and stroke volume low.
Goal: stabilize heart rate, mean pressure, and venous pressure.
Heart Failure Low Type
Low mean pressure, moderately elevated venous pressure, low stroke volume, reduced cardiac output, and low vascular resistance.
Goal: normalize vital signs.
Bleeding
Very low blood volume with reduced filling pressures and increased heart rate. Vascular resistance increases as compensation.
Cure: transfusion plus stable mean pressure above safe levels for several seconds.
Septic Shock
Very low vascular resistance, variable cardiac output, reduced blood volume, low venous pressure, and low mean pressure.
Cure: correct antibiotic followed by stabilization.
THERAPEUTIC MODES
BIPAP in heart failure with adequate pressure: reduces preload, lowers mean pressure, increases heart rate.
Dopamine: inotropic at low doses and vasopressor at higher doses.
Saline fluids: increase blood volume and cardiac output. Reduced effect in heart failure or untreated sepsis.
Furosemide: reduces blood volume, pressure, and congestion.
Nitroglycerin: venous and arterial dilation, reduces workload and mean pressure.
Dobutamine: strong inotrope, increases output and stroke volume, especially when pressure is low.
Norepinephrine: strong vasoconstrictor, increases resistance and venous pressure, may slow heart rate when pressure is high.
Transfusion: essential in bleeding.
Antibiotic: cures sepsis and enables effective use of fluids and vasopressors.
BUTTONS AND CONTROLS
• Monitor toggle with reset.
• Heart button to restart.
• Doctor button to switch character.
• Defibrillator for ventricular arrhythmias.
• Transfusion button to start therapy.
• ECG button to change display.
• Exam mode with ten cases and cumulative score.
• Bottom panel for pause, language, difficulty, instructions, modes, therapies, antibiotic, and ICU mode.
LEGAL NOTICE
SimShockPad is a simplified hemodynamic simulator. It is not an educational or clinical tool. It provides no medical advice. Always consult qualified professionals.}
Read more
Other
Additional Information
| Rating: | |
| Voted: | 7 |
| App Store Link: | |
| Website: | |
| Email: | - |
| Privacy Policy: | |
| Categories: | Medical |
| Size: | 125MB |
| App Age: | 6 months 7 days |
| Release Date: | Jun 09, 2025 |
| Last Update: | Dec 11, 2025 |
| Version: | 6.5 (29) |
Version history
6.5 (29)
Dec 11, 2025
Se ha añadido el idioma japonés tanto en voz como en texto. La versión es la misma que la anterior. No se han realizado otros cambios.
6.3 (28)
Dec 03, 2025
The logic of diagnostic and therapeutic modes has been updated to make it more consistent with real physiology.
A distinction has been added between the right and left circulations:
• The right side is now separated from the left side to increase realism. Pressures are now calculated in a backward direction, from the left ventricle toward the right side, which reflects how congestion usually develops in heart failure.
• New variable: PVR (Pulmonary Vascular Resistance). Similar to SVR in the systemic circuit, a simplified PVR has been introduced.
• PCP (Pulmonary Capillary Pressure or Wedge) is now calculated based on blood volume and left ventricular function.
• PAP (Pulmonary Arterial Pressure) has been updated. PAP is now the sum of the pressure ahead (PCP) plus the resistance of the lungs.
• PVC (Central Venous Pressure) calculation has been revised. PVC now depends on total blood volume and how easily blood can enter the pulmonary artery through PAP.
These changes allow the simulator to show high PCP with normal PVC, enabling the diagnosis of pure left sided failure. This requires specific vasodilator and diuretic therapy instead of treating it as global volume overload. It also allows untreated left sided failure to evolve into right sided failure, as PVC starts to rise minutes later.